Ingrédients

Enhances intestinal iron and zinc absorption; serves as a building block for keratin protein synthesis; may modulate 5-alpha-reductase activity.

Rate-limiting amino acid for keratin synthesis; disulfide bonds from cysteine provide hair shaft structural integrity.

Antifungal with anti-androgenic properties; reduces scalp inflammation and may disrupt local androgen activity.

Potent antioxidant that crosses cell membranes; may inhibit 5-alpha-reductase; reduces NF-kB inflammatory signaling in follicular tissues.

Contains delta-7-stearine which may inhibit 5-alpha-reductase; rich in zinc and phytosterols.

Thymoquinone has anti-inflammatory and antihistamine effects; may modulate telogen effluvium triggers.

Photobiomodulation at 650-670nm wavelength stimulates mitochondrial cytochrome c oxidase, increasing ATP for follicle activity.

Improves microcirculation to scalp; carnosic acid stimulates nerve growth factor for follicle regeneration.

Anti-inflammatory eicosanoids reduce perifollicular inflammation; DHA/EPA support scalp lipid barrier.

Rich in silica (organic silicon) which may strengthen hair shaft; contains antioxidant flavonoids.

EGCG catechins inhibit 5-alpha-reductase and stimulate hair growth via dermal papilla cell proliferation.

Ginsenosides stimulate dermal papilla cell proliferation and inhibit apoptosis; promotes VEGF expression.

Inhibits 5-alpha-reductase, reducing DHT conversion which miniaturizes hair follicles in androgenetic alopecia.

Lignans and polyphenols inhibit 5-alpha-reductase and aromatase; binds sex hormone-binding globulin (SHBG), potentially reducing free DHT at the follicular level.

Inhibits NF-kB and TGF-beta signaling; modulates Wnt/beta-catenin pathway in dermal papilla cells; reduces pro-inflammatory cytokines (TNF-alpha, IL-6) in the perifollicular …

Furostanol saponins (e.g., protodioscin) inhibit 5-alpha-reductase; phytoestrogens may modulate hormonal balance; rich in protein and nicotinic acid for follicle nutrition.

Withanolides modulate HPA axis activity and reduce cortisol; anti-inflammatory effects may support follicle health under chronic stress conditions.

Essential for hemoglobin synthesis; iron deficiency impairs oxygen delivery to hair follicle matrix cells.

Selenoproteins protect hair follicle cells from oxidative damage; essential for thyroid hormone metabolism.

Cofactor for >300 enzymes including those in DNA/RNA synthesis; critical for hair follicle cell proliferation.

Contributes to collagen and keratin cross-linking; supports connective tissue integrity around hair follicles; enhances hair shaft mechanical properties.

Multi-target approach: ashwagandha (stress), saw palmetto (DHT), curcumin (inflammation), tocotrienols (oxidation).

Marine-derived AminoMar complex provides shark cartilage proteins and mollusk powder for hair matrix nourishment.

GHK-Cu stimulates dermal papilla cells, upregulates growth factors (VEGF, FGF), promotes ECM remodeling, and enhances follicle blood supply.

Dual 5-alpha-reductase inhibitor (Type I and II), reducing DHT by ~90%, more potent than finasteride.

Type II 5-alpha-reductase inhibitor reducing serum and scalp DHT by approximately 70%.

Vasodilator that opens potassium channels, increasing blood flow to follicles and prolonging anagen phase.

Androgen receptor antagonist; reduces androgen-mediated follicular miniaturization in female pattern hair loss.

Controlled dermal injury activates wound healing cascade, releasing growth factors and increasing topical absorption.

Platelet-derived growth factors (PDGF, VEGF, EGF) stimulate dermal papilla cells and prolong anagen phase.

Provides proline and glycine amino acids for keratin synthesis; supports dermal papilla extracellular matrix.

Direct structural protein supplementation; solubilized keratin provides bioavailable cysteine for hair shaft.

Penetrates hair follicle, inhibits phosphodiesterase, counteracts testosterone-driven growth suppression.

Organic sulfur donor for keratin cross-linking; may extend anagen phase via anti-inflammatory mechanisms.

Cofactor for carboxylase enzymes essential for keratin synthesis and fatty acid metabolism in hair follicles.

Essential for nucleotide synthesis and methylation reactions in rapidly dividing follicular keratinocytes.

Essential for red blood cell formation and DNA synthesis in rapidly dividing hair follicle cells.

Stimulates hair follicle cycling; vitamin D receptor (VDR) activation is essential for anagen initiation.

Reduces lipid peroxidation in scalp follicles; tocotrienols may inhibit oxidative stress-mediated follicle miniaturization and support microcirculation.

Retinoid receptors (RAR/RXR) in dermal papilla cells regulate hair follicle cycling; vitamin A supports sebaceous gland function and epithelial cell …